- Dec 25, 2016
- 25
- 35
- 94
Sadly we lost one of our hens a few days ago. She was a young 18 months old. 
Their preliminary results were for Hemorrhagic liver disease (fatty liver). I personally feel pretty awful about this because it means there is something we probably could've been doing differently to prevent this.
The small flock are on a standard 16% layer crumble w/ oyster shells available. They occasionally get treats like mealworms - maybe a handful or two a day spread across 4 of them. They self regulate their feed out of a treadle feeder. The hen herself seemed like a healthy weight and didn't appear to be overeating.
They live in an 11x11 run, have a 4x4 coop, and have at least a couple of hours of larger backyard foraging every day.
It seems there are a few ways to try to prevent this, though sometimes the hen can be genetically predisposed?
What would you recommend we do preventatively for the rest of the hens? We can change up feed, do away with treadle feeding, supplement, whatever might help. This was particularly hard because she was fine and happy merely hours earlier and next thing we knew it she was gone. I'm worried others might be in the same condition and asymptomatic.
Thanks so much in advance for your help.
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We submitted this description as part of the necropsy:
Results:

Their preliminary results were for Hemorrhagic liver disease (fatty liver). I personally feel pretty awful about this because it means there is something we probably could've been doing differently to prevent this.
The small flock are on a standard 16% layer crumble w/ oyster shells available. They occasionally get treats like mealworms - maybe a handful or two a day spread across 4 of them. They self regulate their feed out of a treadle feeder. The hen herself seemed like a healthy weight and didn't appear to be overeating.
They live in an 11x11 run, have a 4x4 coop, and have at least a couple of hours of larger backyard foraging every day.
It seems there are a few ways to try to prevent this, though sometimes the hen can be genetically predisposed?
What would you recommend we do preventatively for the rest of the hens? We can change up feed, do away with treadle feeding, supplement, whatever might help. This was particularly hard because she was fine and happy merely hours earlier and next thing we knew it she was gone. I'm worried others might be in the same condition and asymptomatic.
Thanks so much in advance for your help.
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We submitted this description as part of the necropsy:
Hen was behaving normally as of approximately 5:30 pm on April 13. At around 7:15 pm when closing up the run for the night we noticed she was lethargic, standing momentarily but sitting quickly, slightly open beak (breaking normal –to-slow)., not engaging other hens, eyes closing, walking mostly normally with bouts of sitting and wobbling. Separated from the flock (3 other hens) and administered 1.5cc Nutri-Drench Poultry directly. Crop full and soft, maybe watery. Abdomen felt full and round. Treated for egg binding: set hen in warm bath for 15 minutes, dried off and massaged abdomen, left her in bathtub with towels and perch around 8 pm, turned off light for the evening. Hen did not make it through the night. Feces was minimal, white, viscous (like glue).
Egg production seemed normal.
Nutrition: Layer feed, yard foraging, oyster shell available.
Housing: 11x11 run, 4x4 coop (3 other hens), larger backyard for monitored foraging.
Housing: rice straw bedding in run and coop.
Vaccinations: Marek’s
Flock was wormed approximately 3 months ago (Wazine).
Results:
Gross Observations
A female chicken is submitted for postmortem examination in good body condition with an abundant amount of reserve fat and in fair postmortem preservation. Within the coelomic cavity there is a large amount of clotted blood that is adhered to a ruptured liver capsule. The liver is expanded by dissecting hemorrhage which forms a hematoma within the capsule of the liver. No other significant gross findings are observed.
Case Summary
The gross findings of hemorrhage within the coelom and also within the liver are consistent with a diagnosis of hemorrhagic liver disease. The cause of this disease is not completely understood, but it is a very common disease that is seen in backyard chickens. Rupture of the liver and the liver capsule leads to exsanguination and death. It has been assocaited with chickens that are fed a high concentrate diet and have an overabundance of reserve fat. Histopathologic examination is currently pending and additional reports will follow as significant test results are updated.