See also
and this
also this
Yeah, why not.
In my (admittedly limited) experience, the "alternate" opinions on this come mostly either from those who understand the risks but choose to do so anyways [like myself] and those who lack the experience and knowledge to recognize the damage for what it is, particularly since sudden death by kidney failure isn't something most BYCers would trace to excess calcium, most of us don't get all up inside our birds to have a periodic look around, and the visually obvious long term symptoms - like gout - are often associated with old age, rather than an underlying pathology. There are a lot of the latter. The science on this is well settled.
But if you don't keep your roos long, the risk is very low. If "good enough" is good enough - you don't care that your birds could have been a little bigger under a more optimum diet - for you, then it doesn't matter. and again, for birds harvested young, risks are quite low. If you use a bifurcated feeding regimen, such that your birds are on a starter/grower or all flock-type formulation prior to point of lay (that is, the critical time period when they are most susceptible to excess calcium), again, your risks are quite low [this is basically what I do]. If your birds free range a varied environment, that helps. Calcium sources other than calcium carbonate helps. Additional Phosphorus (to a point) helps. There are vitamins that help regulate CA / P Balance which can also buffer the effect of excess dietary calcium.
and this
also this
Yeah, why not.
In my (admittedly limited) experience, the "alternate" opinions on this come mostly either from those who understand the risks but choose to do so anyways [like myself] and those who lack the experience and knowledge to recognize the damage for what it is, particularly since sudden death by kidney failure isn't something most BYCers would trace to excess calcium, most of us don't get all up inside our birds to have a periodic look around, and the visually obvious long term symptoms - like gout - are often associated with old age, rather than an underlying pathology. There are a lot of the latter. The science on this is well settled.
But if you don't keep your roos long, the risk is very low. If "good enough" is good enough - you don't care that your birds could have been a little bigger under a more optimum diet - for you, then it doesn't matter. and again, for birds harvested young, risks are quite low. If you use a bifurcated feeding regimen, such that your birds are on a starter/grower or all flock-type formulation prior to point of lay (that is, the critical time period when they are most susceptible to excess calcium), again, your risks are quite low [this is basically what I do]. If your birds free range a varied environment, that helps. Calcium sources other than calcium carbonate helps. Additional Phosphorus (to a point) helps. There are vitamins that help regulate CA / P Balance which can also buffer the effect of excess dietary calcium.
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